参考文献/References:
[1] Chalasani N, Younossi Z, Lavine JE, et al. The diagnosis and management of nonalcoholic fatty liver disease:practice guidance from the American Association for the Study of Liver Diseases[J].Hepatology,2018,67(1):328-357.DOI:10.1002/hep.29367.
[2] Lau JK, Zhang X, Yu J. Animal models of non-alcoholic fatty liver disease: current perspectives and recent advances[J].J Pathol, 2017,241(1):36-44.DOI:10.1002/path.4829.
[3] Fan M, Choi YJ, Tang Y, et al. Efficacy and mechanism of polymerized anthocyanin from grape-skin extract on high-fat-diet-induced nonalcoholic fatty liver disease[J].Nutrients,2019,11(11):2586.DOI:10.3390/nu11112586.
[4] Chen S, Zhao X, Wan J, et al. Dihydromyricetin improves glucose and lipid metabolism and exerts anti-inflammatory effects in nonalcoholic fatty liver disease:a randomized controlled trial[J].Pharmacol Res, 2015,99(1):74-81.DOI:10.1016/j.phrs.2015.05.009.
[5] Le L, Jiang B, Wan W, et al. Metabolomics reveals the protective of dihydromyricetin on glucose homeostasis by enhancing insulin sensitivity[J]. Sci Rep, 2016,6(1):36184.DOI:10.1038/srep36184.
[6] Sidossis L, Kajimura S. Brown and beige fat in humans: thermogenic adipocytes that control energy and glucose homeostasis[J].J Clin Invest,2015,125(2):478-486.DOI:10.1172/JCI78362.
[7] Singh AK, Aryal B, Chaube B, et al. Brown adipose tissue derived ANGPTL4 controls glucose and lipid metabolism and regulates thermogenesis[J].Mol Metab,2018,11:59-69.DOI:10.1016/j.molmet.2018.03.011.
[8] Liu W, Cao H, Yan J, et al. ‘Micro-managers' of hepatic lipid metabolism and NAFLD[J].Wiley Interdiscip Rev RNA, 2015,6(5):581-593.DOI:10.1002/wrna.1295.
[9] Kawano Y, Cohen DE. Mechanisms of hepatic triglyceride accumulation in non-alcoholic fatty liver disease[J].J Gastroenterol,2013,48(4):434-441.DOI:10.1007/s00535-013-0758-5.
[10] Frederico MJ, Vitto MF, Cesconetto PA, et al. Short-term inhibition of SREBP-1c expression reverses diet-induced non-alcoholic fatty liver disease in mice[J].Scand J Gastroenterol,2011,46(11):1381-1388.DOI:10.3109/00365521.2011.613945.
[11] Zhang M, Sun W, Zhou M, et al. MicroRNA-27a regulates hepatic lipid metabolism and alleviates NAFLD via repressing FAS and SCD1[J].Sci Rep, 2017,7(1):14493.DOI:10.1038/s41598-017-15141-x.
[12] Linden AG, Li S, Choi HY, et al. Interplay between ChREBP and SREBP-1c coordinates postprandial glycolysis and lipogenesis in livers of mice[J].J Lipid Res, 2018,59(3):475-487.DOI:10.1194/jlr.M081836.
[13] Wang C, Yan L, Hao M, et al. Astragaloside Ⅳ inhibits triglyceride accumulation in insulin-resistant HepG2 cells via AMPK-induced SREBP-1c phosphorylation[J].Front Pharmacol,2018,9(1):345.DOI:10.3389/fphar.2018.00345.
[14] Jung YA, Kim HK, Bae KH, et al. Cilostazol inhibits insulin-stimulated expression of sterol regulatory binding protein-1c via inhibition of LXR and Sp1[J].Exp Mol Med,2014,46(1):e73.DOI:10.1038/emm.2013.143.
[15] Wang GX, Zhao XY, Meng ZX, et al. The brown fat-enriched secreted factor Nrg4 preserves metabolic homeostasis through attenuation of hepatic lipogenesis[J].Nat Med,2014,20(12):1436-1443.DOI:10.1038/nm.3713.
[16] Jung EJ, Kwon SW, Jung BH, et al. Role of the AMPK/SREBP-1 pathway in the development of orotic acid-induced fatty liver[J].J Lipid Res,2011,52(9):1617-1625.DOI: 10.1194/jlr.M015263.
[17] Shi LY, Zhang T, Liang XY, et al. Dihydromyricetin improves skeletal muscle insulin resistance by inducing autophagy via the AMPK signaling pathway[J].Mol Cell Endocrinol,2015,409:92-102.DOI:10.1016/j.mce.2015.03.009.
[18] Guo L,Zhang HF,Yan XP. Protective effect of dihydromyricetin revents fatty liver through nuclear factor κB/p53/B cell lymphoma 2 associated X protein signaling pathways in a rat model[J].Mol Med Rep,2019,19(3):1638-1644.DOI:10.3892/mmr.2018.9783.
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