参考文献/References:
[1] Köhling HL,Plummer SF,Marchesi JR,et al. The microbiota and autoimmunity:their role in thyroid autoimmune diseases[J].Clin Immunol,2017,183:63-74.DOI:10.1016/j.clim.2017.07.001.
[2] Ishaq HM,Mohammad IS,Guo H,et al.Molecular estimation of alteration in intestinal microbial composition in Hashimoto's thyroiditis patients[J].Biomed Pharmacother,2017,95:865-874.DOI:10.1016/j.biopha.2017.08.101.
[3] Lahner E,Conti L,Cicone F,et al.Thyro-entero-gastric autoimmunity:pathophysiology and implications for patient management[J].Best Pract Res Clin Endocrinol Metab,2020,34(1):101373.DOI:10.1016/j.beem.2019.101373.
[4] Sterzl I,Hrdá P,Matucha P,et al.Anti-helicobacter pylori,anti-thyroid peroxidase,anti-thyroglobulin and anti-gastric parietal cells antibodies in Czech population[J].Physiol Res,2008,57:S135-S141.DOI:10.33549/physiolres.931498.
[5] Xue Y,Enosi Tuipulotu D,Tan WH,et al.Emerging activators and regulators of inflammasomes and pyroptosis[J].Trends Immunol,2019,40(11):1035-1052.DOI:10.1016/j.it.2019.09.005.
[6] Guo Q,Wu Y,Hou Y,et al.Cytokine secretion and pyroptosis of thyroid follicular cells mediated by enhanced NLRP3,NLRP1,NLRC4,and AIM2 inflammasomes are associated with autoimmune thyroiditis[J].Front Immunol,2018,9:1197.DOI:10.3389/fimmu.2018.01197.
[7] Liu J,Mao C,Dong L,et al.Excessive iodine promotes pyroptosis of thyroid follicular epithelial cells in Hashimoto's thyroiditis through the ROS-NF-kappa B-NLRP3 pathway[J].Front Endocrinol(Lausanne),2019,10:778.DOI:10.3389/fendo.2019.00778.
[8] Liu X,Bai X,Zhao J,et al.Associations between NLRC4 gene polymorphisms and autoimmune thyroid disease[J].Biomed Res Int,2020,2020:1378427.DOI:10.1155/2020/1378427.
[9] Yang PC,Li XJ,Yang YH,et al.The influence of bifidobacterium bifidum and bacteroides fragilis on enteric glial cell-derived neurotrophic factors and inflammasome[J].Inflammation,2020,43(6):2166-2177.DOI:10.1007/s10753-020-01284-z.
[10] Seo SU,Kamada N,Muñoz-Planillo R,et al.Distinct commensals induce interleukin-1 beta via NLRP3 inflammasome in inflammatory monocytes to promote intestinal inflammation in response to injury[J].Immunity,2015,42(4):744-755.DOI:10.1016/j.immuni.2015.03.004.
[11] Vierbuchen T,Bang C,Rosigkeit H,et al.The human-associated archaeon Methanosphaera stadtmanae is recognized through its RNA and induces TLR8-dependent NLRP3 inflammasome activation[J].Front Immunol,2017,8:1535.DOI:10.3389/fimmu.2017.01535.
[12] Macia L,Tan J,Vieira AT,et al.Metabolite-sensing receptors GPR43 and GPR109A facilitate dietary fibre-induced gut homeostasis through regulation of the inflammasome[J].Nature Commun,2015,6:6734.DOI:10.1038/ncomms7734.
[13] Ratajczak W,Ry A,Mizerski A,et al.Immunomodulatory potential of gut microbiome-derived short-chain fatty acids(SCFAs)[J].Acta Biochim Pol,2019,66(1):1-12.DOI:10.18388/abp.2018_2648.
[14] Xu M,Jiang Z,Wang C,et al.Acetate attenuates inflammasome activation through GPR43-mediated Ca2+-dependent NLRP3 ubiquitination[J].Exp Mol Med,2019,51(7):1-13.DOI:10.1038/s12276-019-0276-5.
[15] Yao X,Zhang C,Xing Y,et al.Remodelling of the gut microbiota by hyperactive NLRP3 induces regulatory T cells to maintain homeostasis[J].Nature Commun,2017,8(1):1896.DOI:10.1038/s41467-017-01917-2.
[16] Tye H,Yu CH,Simms LA,et al.NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease[J].Nat Commun,2018,9(1):3728.DOI:10.1038/s41467-018-06125-0.
[17] Sellin ME,Müller AA,Felmy B,et al.Epithelium-intrinsic NAIP/NLRC4 inflammasome drives infected enterocyte expulsion to restrict salmonella replication in the intestinal mucosa[J].Cell Host Microbe,2014,16(2):237-248.DOI:10.1016/j.chom.2014.07.001.
[18] Yin J,Sheng B,Yang K,et al.The protective roles of NLRP6 in intestinal epithelial cells[J].Cell Prolif,2019,52(2):e12555.DOI:10.1111/cpr.12555.
[19] Fitzpatrick Z,Frazer G,Ferro A,et al.Gut-educated IgA plasma cells defend the meningeal venous sinuses[J].Nature,2020,587(7834):472-476.DOI:10.1038/s41586-020-2886-4.
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