参考文献/References:
[1] Saeedi P,Petersohn I,Salpea P,et al.Global and regional diabetes prevalence estimates for 2019 and projections for 2030 and 2045:results from the International Diabetes Federation Diabetes Atlas,9th edition[J].Diabetes Res Clin Pract,2019,157:107843.DOI:10.1016/j.diabres.2019.107843.
[2] Thapa D,Xie B,Zhang M,et al.Adropin treatment restores cardiac glucose oxidation in pre-diabetic obese mice[J].J Mol Cell Cardiol,2019,129:174-178.DOI:10.1016/j.yjmcc.2019.02.012.
[3] Lakshmanan AP,Harima M,Sukumaran V,et al.Modulation of AT-1R/AMPK-MAPK cascade plays crucial role for the pathogenesis of diabetic cardiomyopathy in transgenic type 2 diabetic(Spontaneous Diabetic Torii)rats[J].Biochem Pharmacol,2012,83(5):653-660.DOI:10.1016 /j.bcp.2011.11.018.
[4] Zhang Z,Zhang L,Zhou Y,et al.Increase in HDAC9 suppresses myoblast differentiation via epigenetic regulation of autophagy in hypoxia[J].Cell Death Dis,2019,10(8):552.DOI:10.1038/s41419-019-1763-2.
[5] Zhang J,Zhang Y,Mo F,et al.The roles of HIF-1α in radiosensitivity and radiation-induced bystander effects under hypoxia[J].Front Cell Dev Biol,2021,9:637454.DOI:10.3389/fcell.2021.637454.
[6] Ruze A,Chen BD,Liu F,et al.Macrophage migration inhibitory factor plays an essential role in ischemic preconditioning-mediated cardioprotection[J].Clin Sci(Lond),2019,133(5):665-680.DOI:10.1042/CS20181013.
[7] Karwi QG,Wagg CS,Altamimi TR,et al.Insulin directly stimulates mitochondrial glucose oxidation in the heart[J].Cardiovasc Diabetol,2020,19(1):207.DOI:10.1186/s12933-020-01177-3.
[8] Al-Damry NT,Attia HA,Al-Rasheed NM,et al.Sitagliptin attenuates myocardial apoptosis via activating LKB-1/AMPK/Akt pathway and suppressing the activity of GSK-3β and p38α/MAPK in a rat model of diabetic cardiomyopathy[J].Biomed Pharmacother,2018,107:347-358.DOI:10.1016/j.biopha.2018.07.126.
[9] De Nicola GF,Bassi R,Nichols C,et al.The TAB1-p38α complex aggravates myocardial injury and can be targeted by small molecules[J].JCI Insight,2018,3(16):e121144.DOI:10.1172/jci.insight.121144.
[10] Shlomai G,Zelenko Z,Antoniou IM,et al.OP449 inhibits breast cancer growth without adverse metabolic effects[J].Endocr Relat Cancer,2017,24(10):519-529.DOI:10.1530/ERC-17-0077.
[11] Serebrovska TV,Portnychenko AG,Portnichenko VI,et al.Effects of intermittent hypoxia training on leukocyte pyruvate dehydrogenase kinase 1(PDK-1)mRNA expression and blood insulin level in prediabetes patients[J].Eur J Appl Physiol,2019,119(3):813-823.DOI:10.1007/s00421-019-04072-2.
[12] Syafril S,Lindarto D,Lelo A,et al.The effect of puguntano leaf extract(curanga fel-terrae merr.)on P38 mapk levels and glut-4 expression in type 2 diabetic rat muscle[J].Open Access Maced J Med Sci,2019,7(4):521-525.DOI:10.3889/oamjms.2019.165.
[13] Pentassuglia L,Heim P,Lebboukh S,et al.Neuregulin1β promotes glucose uptake via PI3K/Akt in neonatal rat cardiomyocytes[J].Am J Physiol Endocrinol Metab,2016,310(9):E782-E794.DOI:10.1152/ajpendo.00259.2015.
[14] Wang C,Zhu L,Yuan W,et al.Diabetes aggravates myocardial ischaemia reperfusion injury via activating Nox2-related programmed cell death in an AMPK-dependent manner[J].J Cell Mol Med,2020,24(12):6670-6679.DOI:10.1111/jcmm.15318.
[15] He L,Gomes AP,Xin W,et al.mTORC1 promotes metabolic reprogramming by the suppression of GSK3-dependent Foxk1 phosphorylation[J].Mol Cell,2018,70(5):949-960.e4.DOI:10.1016/j.molcel.2018.04.024.
[16] Kushi R,Hirota Y,Ogawa W.Insulin resistance and exaggerated insulin sensitivity triggered by single-gene mutations in the insulin signaling pathway[J].Diabetol Int,2021,12(1):62-67.DOI:10.1007/s13340-020-00455-5.
[17] Cheng Y,Sun F,Wang L,et al.Virus-induced p38 MAPK activation facilitates viral infection[J].Theranostics,2020,10(26):12223-12240.DOI:10.7150/thno.50992.
[18] Chen F,Chen D,Zhao X,et al.Interleukin-6 deficiency facilitates myocardial dysfunction during high fat diet-induced obesity by promoting lipotoxicity and inflammation[J].Biochim Biophys Acta Mol Basis Dis,2017,1863(12):3128-3141.DOI:10.1016/j.bbadis.2017.08.022.
[19] Qi Y,Xu Z,Zhu Q,et al.Myocardial loss of IRS1 and IRS2 causes heart failure and is controlled by p38α MAPK during insulin resistance[J].Diabetes,2013,62(11):3887-3900.DOI:10.2337/db13-0095.
[20] widerska E,Strycharz J,Wróblewski A,et al.Chronic and intermittent hyperglycemia modulates expression of key molecules of PI3K/Akt pathway in differentiating human visceral adipocytes[J]. Int J Mol Sci,2021,22(14):7712.DOI:10.3390/ijms22147712.
相似文献/References:
[1]项芬芬,张学梅,高英慧,等.p53与物质能量代谢的关系[J].国际内分泌代谢杂志,2017,37(04):262.
Xiang Fenfen*,Zhang Xuemei,Gao Yinghui,et al.Relationship between p53 and material, energy metabolism[J].International Journal of Endocrinology and Metabolism,2017,37(04):262.
[2]金实,赵玉岩,都健.骨钙素及其受体对能量代谢的调节机制[J].国际内分泌代谢杂志,2017,37(06):390.
Jin Shi*,Zhao Yuyan,Du Jian..Regulatory mechanism for energy metabolism by osteocalcin and its receptors[J].International Journal of Endocrinology and Metabolism,2017,37(04):390.
[3]张娴 陈宏.新型脂肪因子与肥胖相关代谢性疾病[J].国际内分泌代谢杂志,2018,38(03):171.[doi:10.3760/cma.j.issn.1673-4157.2018.03.007]
Zhang Xian,Chen Hong.Novel adipokines and obesity-related metabolic diseases[J].International Journal of Endocrinology and Metabolism,2018,38(04):171.[doi:10.3760/cma.j.issn.1673-4157.2018.03.007]
[4]王佳蓓 陈风 刘莹 王涤非.Nur77与物质能量代谢的关系[J].国际内分泌代谢杂志,2019,39(01):49.[doi:10.3760/cma.j.issn.1673-4157.2019.01.012]
Wang Jiabei,Chen Feng,Liu Ying,et al.Relationship between Nur77 and meterial, energy metabolism[J].International Journal of Endocrinology and Metabolism,2019,39(04):49.[doi:10.3760/cma.j.issn.1673-4157.2019.01.012]