[1]张洁,李慧,张献博,等.以重度低钾致横纹肌溶解为首要临床表现的 原发性醛固酮增多症1例[J].国际内分泌代谢杂志,2017,37(05):358-360.
 Zhang Jie,Li Hui,Zhang Xianbo,et al.The first clinical manifestation of rhabdomyolysis due to severe hypokalemic of a patient with primary aldosteronism[J].International Journal of Endocrinology and Metabolism,2017,37(05):358-360.
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以重度低钾致横纹肌溶解为首要临床表现的 原发性醛固酮增多症1例()
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《国际内分泌代谢杂志》[ISSN:1673-4157/CN:12-1383/R]

卷:
37
期数:
2017年05期
页码:
358-360
栏目:
病例报告
出版日期:
2017-09-20

文章信息/Info

Title:
The first clinical manifestation of rhabdomyolysis due to severe hypokalemic of a patient with primary aldosteronism
作者:
张洁李慧张献博郭立新
100730 北京医院内分泌科,国家老年医学中心
Author(s):
Zhang Jie Li Hui Zhang Xianbo Guo Lixin.
Department of Endocrinology, Beijing Hospital, National Center of Gerontology, Beijing 100730, China
文献标志码:
A

参考文献/References:

[1] Zuidema PJ.Cirrhosis and disseminated calcification of the pancreas in patients with malnutrition[J]. Trop Geogr Med, 1959,11(1):70-74.
[2] Mohan V, Nagalotimath SJ, Yajnik CS, et al. Fibrocalculous pancreatic diabetes [J]. Diabetes Metab Rev, 1998,14(2):153-170.
[3] Hassan Z, MohanV, Ali L, et al. SPINK1 is a susceptibility gene for fibrocalculous pancreatic diabetes in subjects from the Indian subcontinent[J]. Am J Hum Genet, 2002,71(4):964-968. DOI:10.1086/342731.
[4] Chowdhury ZM, MeDermott MF, Davey S, et al. Genetic susceptibility to fibrocalculous pancreatic diabetes in Bangladeshi subjects: a family study [J]. Genes Immun, 2002,3(1):5-8. DOI:10.1038/sj.gene.6363814.
[5] Ewald N, Kaufmann C, Raspe A,et al. Prevalence of diabetes mellitus secondary to pancreatic diseases(type 3c)[J]. Diabetes Metab Res Rev,2012,28(4):338-342. DOI:10.1002/dmrr.2260.
[6] Govindarajan M, Mohan V, Deepa R, et al. Histopathology and immunohistochemistry of pancreatic islets in fibrocalculous pancreatic diabetes[J]. Diabetes Res Clin Pract,2001,51(1):29-38.
[7] Maisonneuve P, Lowenfels AB, Bueno-de-Mesquita HB, et al. Past medical history and pancreatic cancer risk: results from a multicenter case-control study[J]. Ann Epidemiol, 2010,20(2):92-98. DOI: 10.1016/j.annepidem.2009.11.010.
[8] Knop FK, Vilsbøll T, Larsen S, et al. Increased postprandial responses of GLP-1 and GIP in patients with chronic pancreatitis and steatorrhea following pancreatic enzyme substitution[J]. Am J Physiol Endocrinol Metab,2007,292(1):E324-E330.DOI:10.1152/ajpendo.00059.2006.
[9] Kanta Barman K, Padmanabhan M, Premalatha G, et al. Prevalence of diabetic complication in fivrocalculous pancreatic diabetic patients and type 2 diabetic patients: a cross-sectional comparative study[J]. J Diabetes Complications, 2004, 18(5): 264-270. DOI: 10.1016 /S1056-8727(03)00074-6.
[10] Mohan V, Premalatha G, Padma A, et al. Fibrocalculous pancreatic diabetes. Long-term survival analysis[J]. Diabetes Care, 1996,19(11):1274-1278.
[11] Chari ST, Mohan V, Pitchumoni CS, et al. Risk of pancreatic carcinoma in tropical calcifying pancreatitis: an epidemiological study[J]. Pancreas, 1994,9(1):62-66.
[12] Mittal N, Mehrotra R, Agarwal G,et al. The clinical spectrum of fibrocalculous pancreatic diabetes in north India[J]. Natl Med J India, 2002,15(6):327-331.

备注/Memo

备注/Memo:
通信作者:郭立新,Email:glx1218@163.com
更新日期/Last Update: 2017-09-30